We observed a substantial increase in glucose uptake (p < 0.05; Figure 5E,F) and tumor growth (p < 0.001; Figure 5G–I) in Ishikawa xenografts with ectopic expression of GAL9; while MGAT4A inhibition reversed these effects (p < 0.05; Figure 5E–I). This evidence concerns the gene MGAT4A and neoplasm.