Glucose transporters are membrane proteins that facilitate glucose across the plasma membrane, which have been reported with N‐glycan modification.[37] Disruption of MGAT4A in murine models has been linked to hyperglycemia and anomalous activities of GLUT2 in pancreatic cells.[38] Thus, we employed tunicamycin, an inhibitor of N‐linked glycosylation,[39] to ascertain the N‐glycosylation status of GLUT1 in EC cells. The gene discussed is SLC2A2; the disease is Hyperglycemia.