FGF23 and Acute hepatitis: However, altogether, our observations already appear to support our hypothesis of a link between FGF23 overproduction and hypophosphatemia, since (i) the complete etiological work-up did not reveal any other plausible causes, (ii) our results were supported by increased serum bio-intact FGF23 levels and the parallel favorable evolution of hepatic and biochemical parameters, and (iii) the liver expression of FGF23 was clearly induced in mouse models of acute hepatitis.