The heatmap shown in Figure 7 emphasizes that AML patients in Group A (showing high PFKM/low MAP1LC3B/low BNIP3 expression and poor prognosis) were characterized by the upregulation of a range of transcripts involved in oncogenic pathways, including glucose transporters (SLC2A1, SLC2A4), lactate/pyruvate transporters (SLC16A1), amino acids transporters (SLC6A9, SLC15A10), positive cell cycle regulation (CCNB1, CCNB2, CCNA2), and the biosynthesis of cofactors (NMNAT3, GCLM, HMBS). Here, MAP1LC3B is linked to acute myeloid leukemia.