CircANKS1B was upregulated in triple-negative breast cancer and acted as a miR-148a-3p and miR-152-3p sponge to increase the expression of transcription factors USF1 and TGF-β1 to activate the TGF-β1/Smad signaling pathway and epithelial-to-mesenchymal transition (EMT) [39]. The gene discussed is USF1; the disease is triple-negative breast carcinoma.