MiR-574 promotes the survival, growth, and migration of different thyroid cancer cell lines and favors tumor formation, as well as cancer-related angiogenesis in xenograft models, through the inhibition of BNIP3/AIF, which synergistically cooperate to activate proapoptotic pathways and the upregulation of MMP-2, MMP-9, and VEGF-A expression, which mediate the prometastatic behavior of thyroid carcinoma cells [66]. Here, VEGFA is linked to thyroid gland carcinoma.