Genes downregulated during early-stage infection have roles in transcription and translation (transcription regulator, 40S ribosomal protein S26, and RS27A protein), polyamine regulation (ornithine decarboxylase antizyme domain-containing protein [47]), protein folding as chaperones (the groes-like protein and HSP20-like chaperone), and cell development/signaling (cell division control/GTP-binding protein). This evidence concerns the gene RPS26 and infection.