Dysregulated inflammation is another risk factor for both CVD and dyslipidaemia in HIV-positive patients.7 This is supported by evidence, including (1) the increased level of high-sensitivity C-reactive protein as a nonspecific inflammatory marker that increases the CVD risk and dyslipidaemia, (2) the increased levels of D-dimer that can indicate a positive association with lipid indicators in patients with coronary artery disease, and (3) the increased production of interleukin-6 that occurs during HIV replication and is associated with dyslipidaemia and increased CVD risk.7 This evidence concerns the gene CRP and inherited lipid metabolism disorder.