SLITRK4 and neoplasm: Recently, Zhou et al., found that the interaction between SLTRK4 and canopy FGF signaling regulator 3 (CNPY3) could enhance the TrkB‐related signaling pathway in gastric cancer metastasis.[8] Metastasis is a multistep process mediated by key genes regulating the complex cell‐extrinsic interaction with surrounding stromal cells, but the role of SLITRK4 in the tumor microenvironment (TME) is still unknown.