Interferon α (IFNα) is a key component of inflammation during the early stages of T1DM,[6] while the pro‐inflammatory cytokines IL1β (Interleukin 1β) and IFNγ (Interferon γ) released by infiltrated mononuclear cells in pancreatic islets are involved later in the development of the disease.[7] Various studies describe islet infiltration by macrophages and elevated levels of cytokines, including IL1β, in type 2 diabetes.[8, 9, 10]. The gene discussed is IL1B; the disease is type 1 diabetes mellitus.