SQOR and familial dilated cardiomyopathy: H2S contributes to electron generation via oxidation by sulfide quinone oxidoreductase (SQR), facilitating electron transport in the mitochondrial respiratory chain and subsequent ATP synthesis.[40] Despite these findings, the precise mechanisms by which H2S influences mitochondrial function in DCM, particularly regarding its role in Fe–S cluster synthesis and mitochondrial respiration, remain unclear.