None. The authors stated that additional research was needed to better understand the pathogenesis of atopic dermatitis, and suggested that future research will likely focus on the modulation or inhibition of certain cytokines, particularly IL-4, IL-13, IL-17, IL-31, and JAK-signal transducer and activator of transcription (STAT) inhibition. The gene discussed is IL17A; the disease is atopic eczema.