ACTA1 and hyperuricemia: To further explore the origin of lumican in the infarcted area in hyperuricemia-related MI, relative lumican expression was characterized in fibroblasts (marked by α-SMA), macrophages (marked by CD68), neutrophils (marked by Ly6G), endothelial cells (marked by CD31), and cardiomyocytes (marked by c-TnT).