ELAVL1 and neoplasm: The collaborative effect of DTX/Lip-HAR and HuR CRISPR/SLN-HAR triggered apoptosis in a significantly higher number of tumor cells than any other treatment, as evidenced in Fig. 8E. Importantly, this potent antitumor effect was achieved without causing any significant differences in the body weights of the tumor-bearing mice across all groups (Fig. 8C).