Overall, the analysis included 48 women, aged 14 to 38 years, affected by various types of lipodystrophy: 27 cases of familial partial lipodystrophy type 2 (Dunnigan type related to LMNA gene) [21, 22, 25, 26, 28-30]; 15 cases of FPLD type 3 [peroxisome proliferator-activated receptors (PPARγ) mutation) [19, 24, 27, 30]; 4 cases of partial lipodystrophy of unknown genetic cause [30]; 3 cases of generalized lipodystrophy, of whom 1 was acquired [20] and 2 congenital (AGPAT2 gene mutation) [23, 30]. This evidence concerns the gene LMNA and familial partial lipodystrophy, Dunnigan type.