In addition, they had a secretome consistent with the tumor scenario.[43] Their cytokine profile indicated a high capacity for migration (EGF), proliferation, intense immune modulation (IL‐13, IL‐12p40, IL‐5, and IL‐10) and activation (MIP‐1 β, MIG, MCP‐3, MCP‐1, IP‐10, IL‐17A, IFN‐α 2, and IFN‐γ) as well as signs of tumor resistance behavior (IL‐5, IL‐8). The gene discussed is CXCL9; the disease is neoplasm.