Our findings (Supplementary Table S2), which are similar to those of previous studies (Mulvihill et al., 2009; Allister et al., 2008), suggest that genetic variants of APOB, microsomal triglyceride transfer protein, LDLR, and 3-hydroxy-3-methylglutaryl-CoA reductase in naringenin-LM are associated with the development of hyperlipidemia (Supplementary Figure S3 and Supplementary Table S3). The gene discussed is LDLR; the disease is hyperlipidemia.