Instead, evidence suggests that CLC-P/Gal10 granules may be externalized to sites of inflammation (Fig. 5a–c), possibly via extracellular vesicles (EVs) or eosinophil extracellular traps (EETs).58, 59, 60 Accordingly, the time-lapse imaging reveals that Dif-EOL1 degranulate CFSE-labelled components into M14K mesothelioma cells (Fig. 3a). The gene discussed is CLC; the disease is mesothelioma.