PRL and metabolic dysfunction-associated steatotic liver disease: Results analyzed through enrichment showed that the signaling pathways for IL-17, Toll-like receptors, Folate biosynthesis, ECM-receptor interaction, Endocrine resistance, Human papillomavirus infection, nonalcoholic fatty liver disease, Mitophagy – animal, Prolactin signaling pathway, Phagosome, PI3K-Akt and TNF signaling pathways, respectively, etc are intimately linked to the development of CP.