While inflammation is acknowledged as a significant contributor to DN development, it is essential to note that DN is not a classic inflammatory disease, as outlined in numerous publications.[8, 10] To further substantiate the regulatory role of evf‐2 in inflammation, we employed an LPS‐induced mouse model using podocyte‐specific evf‐2 KO mice. The gene discussed is DLX6-AS1; the disease is liver dysplastic nodule.