Furthermore, hypoxic conditions lead to the upregulation of aspartyl-tRNA synthetase 1 antisense 1 (DARS-AS1), which recruits METTL3 and METTL14 to mediate m6A modifications that increase the translation of DARS, promoting the expression of ATG5 and ATG3 and thus facilitating protective autophagy in cervical cancer cells [34]. Here, ATG5 is linked to cervical carcinoma.