To further determine the pathogenesis of age-related cataracts, future experiments will need to be done to determine the expression levels of activated Akt, RSK and PKA to determine if there are any age-related or genotype-related expression changes and whether there is any link between the pathological and physiological functions of non-canonical EphA2 signaling. The gene discussed is AKT1; the disease is Age-related cataract.