These included the chemokines eotaxin, KC, MCP-1, MIP-1α, MIP-1β and RANTES; the pro-inflammatory cytokines IFN-γ, IL-1α, IL-6 and IL-17a and the anti-inflammatory cytokine IL-10 in IFNAR−/−-BS, among others. This evidence concerns the gene IFNG and Bloom syndrome.