circRNA generated from cell migration-inducing protein boosts defensive autophagy in prostate cancer cells, leading to increased resistance to anoikis.[32] A different research demonstrated that the activation of catenin beta 1 transcription by nuclear myosin heavy chain 9 resulted in resistance to anoikis and promoted metastasis in gastric cancer cells, both in vitro and in vivo.[33] Consequently, ARGs could serve as prognostic indicators and treatment targets for a range of cancers. Here, CTNNB1 is linked to prostate carcinoma.