Reductions in kidney function (estimated glomerular filtration rate) did not differ between groups, while increases in pre‐injury phase urinary AKI biomarkers (insulin‐like growth factor‐binding protein‐7 (IGFBP7) × tissue inhibitor of metalloproteinases‐2 (TIMP‐2)) were not different between groups and there were no changes in injury phase urinary AKI biomarkers (neutrophil gelatinase‐associated lipocalin (NGAL) and kidney injury molecule‐1 (KIM‐1)). The gene discussed is LCN2; the disease is acute kidney injury.