This study investigated the effect of ATR, which as a redox-active molecule has pro-oxidant capacity and is known to increase ROS levels in a dose-dependent manner, on ferroptosis in breast cancer cells and showed that ATR has the potential to induce ferroptosis in cells by increasing intracellular ROS and lipid ROS levels and decreasing anti-oxidant system-related gene expression levels. Here, ATR is linked to breast cancer.