In gastric cancer cells, 100–400 μM UA induced G0/G1 arrest (in BGC823 cells) or G2/M (in SGC7901 cells) after 24 h treatment and apoptosis with the rise of the Bax:Bcl2 ratio (also in vivo) and caspase 3 activation as well as autophagy. Here, BAX is linked to gastric cancer.