In addition, the lncRNA DRCT was overexpressed in normal cardiac tissues while it was downregulated in patients with DCM; its mechanism of action seems to involve mitochondrial dysfunction; in particular, DCRT inhibited the exon skipping of NDUFS2 (NADH dehydrogenase ubiquinone iron-sulfur protein 2) by binding to PTBP1 (polypyrimidine tract binding protein 1) [49]. The gene discussed is PTBP1; the disease is familial dilated cardiomyopathy.