KRAS and pancreatic ductal adenocarcinoma: reported that LINC00673 encoded novel protein RASON is a key regulator of oncogenic KRAS signaling in pancreatic ductal adenocarcinoma, and could bind directly to KRASG12D/V and inhibit intrinsic and GAP‐mediated GTP hydrolysis, thereby maintaining KRASG12D/V in a highly active state of GTP binding.[53] In this study, we performed an integrative analysis combining transcriptomic and translatomic data, identifying HCP5 as an lncRNA associated with GC that possesses coding potential.