As we did not observe any correlation between surface expression of MR1 allomorphs on cancer cell lines, as measured by flow cytometry, and 7G5.TCR-T efficacy, as measured by IFN-γ release (Figure 2B), we concluded that MR1*04 expression stimulates 7G5.TCR-T to a much greater extent thanMR1*01 when the levels of expression on the cell surface are comparable. This evidence concerns the gene MR1 and cancer.