CeD is characterized by an intestinal Th1 response to dietary gluten, presenting with hypersecretion of proinflammatory proteins in the damaged mucosa or serum, particularly interferon (IFN)-Υ, tumor necrosis factor (TNF), interleukin (IL)-1, and IL-6 (13). The gene discussed is IL6; the disease is cranioectodermal dysplasia.