YTHDF1 and acute myeloid leukemia: In AML, writers (such as METTL3, METTL14, and METTL16), erasers (FTO and ALKBH5), and readers (YTHDF1, YTHDF2, YTHDC1, and IGF2BP2) are all aberrantly upregulated, promoting AML progression.4, 28