Various studies have shown that neuroinflammation in depression models is characterized by the release of toll‐like receptor 4 (TLR‐4), NF‐κB, and proinflammatory cytokines (Gárate et al., 2011; Liu et al., 2021; Zhou et al., 2020). The gene discussed is NFKB1; the disease is depressive symptom measurement.