also mentioned that mice with DM alone lagged in IFN‐γ production but exhibited enhanced Th1 cell activity in combination with TB, which may be related to the increase in late‐stage glycosylation end‐products in patients with DM as well as the increased antigenic stimulation following MTB infection.[104] In addition, unlike the previous studies, Krause et al. This evidence concerns the gene IFNG and diabetes mellitus.