measured the levels of IFN‐γ during anti‐TB treatment in patients.[102] They found that in TB patients with well‐controlled blood glucose who had T2DM, the levels of IFN‐γ secretion returned to levels similar to those of the control group after 6 months of anti‐TB treatment, while the production of IFN‐γ in TB patients with poor glycemic control remained decreased, further confirming the relationship between the inhibition of IFN‐γ secretion and the high blood glucose state.[102] Similarly, the study by Stalenhoef et al. This evidence concerns the gene IFNG and tuberculosis.