ANKRD1 and muscular disease: Increasing evidence has highlighted the significant role of oxidative stress in the pathophysiology of various muscle disorders, both in early and adult‐onset cases,85 In ANKRD1+ myofibers post‐RCT, there was an elevation in NFE2L2, RB1 and ZEB1, suggested a potential promotion of an adaptive antioxidant response during muscle atrophy.