Since neuroinflammation is also thought to contribute to PD pathogenesis, we further exposed the hiPSC‐derived microglia to the physiologically relevant cytokine IFNγ, shown to be elevated in PD brains (Mogi et al., 2007) and primarily secreted by non‐microglial cells (Billiau & Matthys, 2009), and investigated how this affected αSyn processing and clearance. This evidence concerns the gene IFNG and Parkinson disease.