Additionally, MYC expression was found to be elevated in a subset of PDAC.[37, 38] According to a study, Myc is a crucial mediator of the metabolic alterations caused by Kras mutation and is an important driving factor for the survival of pancreatic cancer cells.[39] Another study revealed that, Myc interacts with PIN1 to promote NRF2 expression, which protects pancreatic cancer cells from KRAS‐induced mitochondrial respiratory damage.[40]. The gene discussed is KRAS; the disease is pancreatic neoplasm.