It plays a role in AMI and can serve as a molecular biomarker for screening and diagnosing AMI.[34] The tyrosine kinase Src (cellular-Src) in MI is related to the remodeling of connexin 43 in the border zone myocardial cells after MI.[35] Inhibiting it can improve the levels and conduction velocity of connexin 43 after MI, reduce the induction of arrhythmias, and provide a new approach to alleviate arrhythmias after MI. The gene discussed is GJA1; the disease is cardiac arrhythmia.