Additionally, this data suggested that T‐cell‐mediated antitumor immune responses may be impaired.[34] The ki67 immunohistochemical assay revealed persistent tumor cell proliferation, while TUNEL staining analysis indicated a reduction in apoptosis within the tumor tissues during Anti‐PD1 treatment, suggesting resistance to the current PD1 blockade approach (Figure 1G,H). The gene discussed is MKI67; the disease is neoplasm.