To further investigate the tumor-promoting related roles of the C1 SRSF7+ MCs subtype, we performed enrichment analysis and obtained the upregulated genes DDX5, TPSB2, and CPA3, of which DDX5 interacts with a variety of key pro-tumorigenic molecules and participates in tumorigenic and tumor progression signaling pathways, and when DDX5 is expressed or its post-translational modifications are deregulated, the normal cellular signaling network collapses, leading to many pathological states, including tumorigenesis and tumor progression (52, 70). Here, SRSF7 is linked to neoplasm.