The somatic SNP load per feline ACC was slightly higher than the mutation load reported in human ACCs (median, 17; range, 1–806); however, it should be noted that the range in human ACCs was large.32 Fewer InDels and CNVs were identified in adenomas than in the ACCs; comparable numbers of CNVs are present in human adrenocortical tumors.59 Several CNVs were identified within >1 tumor and affected regions containing genes (including CACNA1H and SLC24A2), which are of interest in human PA.9,60. This evidence concerns the gene SLC24A2 and adenoma.