Our sequencing also identified interesting candidates such as CFL2 (cofilin 2), whose inactivation in nasoparyngeal carcinoma decreased cell proliferation, while apoptosis and radiosensitivity were enhanced [30], or MTDH (metadherin) [31–33], MPRIP (myosin phosphatase Rho interacting protein) [34] and TOB1 (Transducer of ERBB2, 1) [35–37] all involved in response to radiation in cancer cells. The gene discussed is CFL2; the disease is cancer.