SLAMF9 and viral infectious disease: Functional enrichment analysis based on gene ontology revealed that virus-positive Slamf9+ macrophages primarily upregulated genes related to “tissue remodeling” and “vascular endothelial growth factor production”, but not genes associated with cell death-related genes compared to tissue-resident AMs (Supplementary Fig. S3d, e), implying cell death-resistant phenotypes of these macrophages and their potential roles in resolving inflammation and viral infection by pro-fibrosis.