It is well known that pathways such as DNA replication, tumor proliferation signature, G2M checkpoint, p53 pathways, and PI3K/AKT/mTOR signaling pathways are highly correlated with the occurrence and development of cervical cancer (Moody and Laimins, 2010; Bossler et al., 2019; Hussain et al., 2021). This evidence concerns the gene TP53 and cervical carcinoma.