Although LC3 interaction with NLRC5 has been postulated to inhibit HLA-class-I gene expression and contribute to the immune escape of endometrial cancer cells [37], we did not find a significant association of autophagy blockage with chloroquine and the transcriptional activity of HLA-class-I, TAP1, and β2-microglobulin genes. This evidence concerns the gene MAP1LC3A and endometrial cancer.