Dai and collaborators [21] analyzed the biochemical profile and viral load of HBV carriers with chronic infection (dividing them into groups with and without hepatic steatosis) and argued that a decrease in HBV viral load levels and ALT/SGPT, as well as an increase in AST/SGOT and GGT levels, are associated with the development of hepatic fibrosis and advanced hepatic fibrosis. This evidence concerns the gene GOT1 and Hepatic steatosis.