This corroborates what Sun et al. observed by demonstrating that the overexpression of miR-101 reduces the expression of pro-inflammatory cytokines, such as interferon gamma (IFN-γ), interleukin-6 (IL-6), and interleukin-17 (IL-17) A, in systemic lupus erythematosus [36]. Here, IL17A is linked to systemic lupus erythematosus.