The attenuation of renal injury and dysfunction was confirmed by testing the levels of kidney damage-related molecules (increased nephrin and podocin levels, decreased KIM-1—kidney damage-related molecule 1) and biochemical markers of kidney condition (decreased serum creatinine, creatinine clearance, urinary albumin-to-creatinine ratio, urinary albumin excretion ratio). The gene discussed is ALB; the disease is Nephropathy.