However, the results of the studies on this issue are not uniform, as other authors have reported quite contradictory results [8,9,10,11] denying the role of prolactin in endometriosis without tubal occlusion as a factor for the development of infertility and have focused on impaired ovarian reserve and reduced ovarian response in endometriosis, as indicated by elevated FSH and lower anti-Mullerian hormone. The gene discussed is BRD2; the disease is endometriosis.