As shown in Figure S1, our findings indicate that (1) CENPK–delta8 is strongly correlated with AR or AR–V7, consistent with its potential role in resistance to Abiraterone; (2) high CENPK–delta8 expression is significantly correlated with high AR activity score, but not neuroendocrine score (NEPC), while CENPK all-transcripts are reversed; and (3) wild-type CENPK is highly correlated with cell-cycle progression score, a score characterizing proliferative phenotypes of the tumor, while CENPK–delta8 is moderately correlated. This evidence concerns the gene CENPK and neoplasm.