The pathogenesis of PCOS has been linked to an activation of the inflammatory response mediated by C‐reactive protein (CRP) and increased levels of inflammatory cytokines, blood lymphocytes, and monocytes, together with an enhanced expression of adhesion molecules and increased presence of markers of oxidative stress, such as lipid peroxidation and myeloperoxidase (MPO),6, 7 all of which promote the initial stages of atherosclerosis8—the main risk factor for CVD. The gene discussed is MPO; the disease is polycystic ovary syndrome.